上海交通大学内科学专业导师介绍：贾楠

►基本信息导师姓名：贾楠性别：男专业名称：内科学专业代码：100201培养单位：附属瑞金医院专业领域：心血管病导师类型：硕士生导师邮箱地址：ji

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2015-07-09

　　►学术论文

作者	论文标题	期刊名	出版年	卷期页码
Li YQ, Li XB, Guo SJ, Chu SL, Gao PJ, Zhu DL, Niu WQ, Jia N （通讯作者）	Apocynin attenuates oxidative stress and cardiac fibrosis in angiotensin II-induced cardiac diastolic dysfunction in mice	Acta Pharmacologica Sinica	2013	00:00-00:00
Li X, Qi Y, Li Y, Zhang S, Guo S, Chu S, Gao P, Zhu D, Wu Z, Lu L, Shen W, Jia N （通讯作者）, Niu W	The Impact of Mineralocorticoid Receptor Antagonists on Changes in Cardiac Structure and Function of Left Ventricular Dysfunction: A Meta-Analysis of Randomized Controlled Trials	Circulation Heart Failure	2013	00:00-00:00
Jia N （共同第一作者）,Dong P,Ye Y,Qian C,Dai Q	Allopurinol attenuates oxidative stress and cardiac fibrosis in angiotensin II-induced cardiac diastolic dysfunction	Cardiovasc Ther	2012	30(2):117-123
Li X, Li Y, Jia N （通讯作者）, Guo S, Chu S, Niu W	Angiotensin-converting enzyme gene deletion allele increases the risk of left ventricular hypertrophy: evidence from a meta-analysis	Mol Biol Rep	2012	39(12):10063-10075
Li Y, Li X, Jia N （通讯作者）, Guo S, Chu S, Niu W	Meta-analysis of the association between angiotensin II receptor, type 1 gene A1166C polymorphism and coronary artery disease in Chinese populations	J Renin Angiotensin Aldosterone Syst	2012	00:00-00
李晓波,贾楠（通讯作者）	肾动脉肌纤维发育不良的诊断治疗进展	心血管微创医学杂志	2012	1(1):91- 97
P Dong, M Karaayvaz, N Jia （共同第一作者）, M Kaneuchi, J Hamada, H Watari, S Sudo, J Ju and N Sakuragi	Mutant p53 gain-of-function induces epithelial-mesenchymal transition through modulation of the miR-130b-ZEB1 axis	Oncogene	2012	00:00-00
Li X, Li Y, Song B, Guo S, Chu S, Jia N （通讯作者）, Niu W.	Hematopoietically-expressed homeobox gene three widely-evaluated polymorphisms and risk for diabetes: a meta-analysis	PLoS One	2012	7(11):e49917-e49917
李晓波,李玉琼,陶波,贾楠（通讯作者）,初少莉,高平进	肾动脉显像测定的肾小球滤过率对年轻患者肾动脉狭窄诊断和肾功能的评估价值	中华高血压杂志	2012	20(12):1144-1151
贾楠（通讯作者）	肾动脉支架术：随机临床试验与临床实践的矛盾统一	中华医学信息导报	2011	26(18):20-21
叶瑛,贾楠（通讯作者）	肾素-血管紧张素醛固酮系统与肾动脉狭窄	临床内科杂志	2011	28:8-9
李晓波,贾楠（通讯作者）	欧洲心脏病学会2011年关于肾动脉疾病的指南内容	中国全科医学	2011	11:6-9
叶瑛,贾楠（通讯作者）	动脉硬化性肾动脉狭窄相关临床试验的进展与分析	中华高血压杂志	2010	18:1025-1028
叶瑛,贾楠（通讯作者）	高尿酸血症-心血管事件的独立危险因素	国际心血管病杂志	2010	37:229-231
叶瑛,贾楠（通讯作者）	瑞舒伐他汀的临床研究进展	中国新药与临床杂志	2010	:29:813-815
Jia N （通讯作者）, Qiuping H, Wei J, Jianjun Z Qiuyan D, Shaowen L	Cardioprotective effects of granulocyte colony-stimulating factor in Angiotensin-II Induced Cardiac Remodeling	Clin Exp Pharmacol Physiol	2009	36(3):262-266
Dong P, Xu Z, Jia N, Li D, Feng Y	Elevated expression of p53 gain-of-function mutation R175H in endometrial cancer cells can increase the invasive phenotypes by activation of the EGFR/PI3K/AKT pathway	Mol Cancer	2009	16(8):103.
Dong PX, Jia N, Xu ZJ, Liu YT, Li DJ Feng YJ	Silencing of IQGAP1 by shRNA inhibits the invasion of ovarian carcinoma HO-8910PM cells in vitro	J Exp Clin Cancer Res	2008	27(1):77.
贾楠（通讯作者）,黄秋萍,金炜,张建军,戴秋艳,刘少稳	粒细胞集落刺激因子对血管紧张素II所致小鼠心室肥厚的保护作用	中华高血压杂志	2008	16(10):919-923
贾楠（通讯作者）,黄秋萍,金炜,张建军,孙宝贵	奥美沙坦与替莫普利合用对逆转左室肥厚无协同作用	中华高血压杂志	2007	4(15):285-288
贾楠（通讯作者）,黄秋萍,金炜,孙宝贵	奥美沙坦通过减低整合素β5的表达促进心肌肥大的消退	中国心血管杂志	2007	2(12):12-17
Matsui Y, Okamoto H, Jia N, Akino M, Uede T, Kitabatake A, Nishihira J	Blockade of macrophage migration inhibitory factor ameliorates experimental autoimmune myocarditis	Journal of Molecular and Cellular Cardiology	2004	37(2):557-566
Matsui Y, Jia N （共同第一作者）,Okamoto H, Kon S Onozuka H, Akino M, Liu L, Morimoto J, Rittling SR, Denhardt D, Kitabatake A, Uede T	Role of osteopontin in cardiac fibrosis and remodeling in angiotensin II-induced cardiac hypertrophy	Hypertension	2004	43(6):1195-1201.
Matsui Y, Okamoto H, Inobe M, Jia N, Akino M, Sugawara T, Kon S, Uede T	Osteopontin Deficiency Attenuates Atherosclerosis in Female Apolipoprotein E Deficient Mice	Circulation	2003	108(17):IV-21
Matsui Y, Okamoto H, Inobe M, Jia N, Akino M, Sugawara T, Kon S, Uede T	Adenovirus-Mediated Gene Transfer of ICOSIg Fusion Protein Ameliorates Ongoing Experimental Autoimmune Myocarditis.	Circulation	2003	108(17):IV-518
Jia N, Okamoto H, Kon S, Matsui Y, Akino M, Sugawara T, Uede T, Kitabatake A	Osteopontin Is Essential for Cardiac Fibrosis in Angiotensin II-Induced Hypertrophy	Journal of Cardiac Failure	2003	9(5):S146-146
Matsui Y, Rittling SR, Okamoto H, Inobe M, Jia N, Shimizu T, Akino M, Sugawara T, Morimoto J, Kimura C, Kon S, Denhardt D, Kitabatake A, Uede T	Osteopontin Deficiency Attenuates Atherosclerosis in Female Apolipoprotein E-Deficient Mice	Arteriosclerosis thrombosis and vascular biology	2003	23(6):1029-1034
Jia N, Okamoto H, Matsui Y, Sugawara T, Akino M, Kitabatake A	A Newly Developed Angiotensin II Type 1 Receptor Antagonist, CS866, Promotes Regression of Cardiac Hypertrophy by Reducing Beta-1 Integrin Expression	Journal of Cardiac Failure	2003	9(5):S187-187
Matsui Y, Okamoto H, Inobe M, Jia N, Shimizu T, Akino M, Sugawara T, Tezuka K, Nakayama Y, Morimoto J, Kimura C, Kon S, Miyazaki T, Kitabatake A, Uede T	Adenovirus-Mediated Gene Transfer of ICOSIg Fusion Protein Ameliorates Ongoing Experimental Autoimmune Myocarditis	Human gene therapy	2003	14(6):521-532
Jia N, Okamoto H, Shimizu T, Chiba S, Matsui Y, Sugawara T, Akino M, Kitabatake A	A Newly Developed Angiotensin II Type 1 Receptor Antagonist, CS866, Promotes Regression of Cardiac Hypertrophy by Reducing Beta-1 Integrin Expression	Hypertension Research	2003	26:737-742
Shimizu T, Okamoto H, Chiba S, Matsui Y, Sugawara T, Akino M, Jia N, Kumamoto H, Onozuka H, Mikami T, Kitabatake A	VEGF-mediated angiogenesis is impaired by angiotensin type 1 receptor blockade in cardiomyopathic hamster hearts	Cardiovascular research	2003	58(1):203-212
Jia N, Okamoto H, Kitabatake A	Integrin Beta-1 Results in Cardiac Hypertrophy Through Angiotensin II Type 1 Receptor	Journal of Molecular and Cellular Cardiology	2003	34(10):A17
Jia N, Akino M, Okamoto H, Watanabe M, Kitabatake A.	Changing Prognosis in Dilated Cardiomyopathy	Circulation Journal	2002	66 Suppl: 723.
Okamoto H, Jia N, Sugawara T, Matsui Y, Shimizu T, Akino M, Chiba S, Kitabatake A	Changing Prognosis in Japanese Patients with Dilated Cardiomyopathy	Circulation	2002	106(19):751-751

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