暨南大学医学院导师介绍:费嘉
姓名:费嘉 性别:男 职称:教授 学院:医学院 研究方向:(1)小核酸药物筛选与肿瘤基因治疗.,(2)肿瘤非编码
【代表性论文】
1.miR-181a post-transcriptionally downregulates oncogenic RalA and contributes to cell growth inhibition, G2 phase arrest and apoptosis in chronic myelogenous leukemia (CML) cells(submitted, corresponding author)
2.Inhibition of small GTPase RalA regulates growth, cell cycle and arsenic-induced apoptosis in chronic myeloid leukemia(CML) cell. Cellular Signalling(revised)(SCI, IF 4.243, A1, corresponding author)
3.Anti-miR-21 Oligonucleotide Sensitizes Leukemic K562 Cells to Arsenic Trioxide By Inducing Apoptosis. Cancer Science.2010,101(4): 948–954 Epub 2010 Jan 7(SCI, IF 3.846, A1, corresponding author)
4.Antisense oligonucleotide against miR-21 inhibites growth, migration and induces apoptosis in leukemic K562 cells. Leukemia and Lymphoma 2010, 51(4):694-701 Epub 2010 Feb 8. (SCI, IF 2.492, A2, corresponding author )
5.Anti-miR-21 oligonucleotide enhances chemosensitivity of leukemic HL60 cells to arabinosylcytosine by inducing apoptosis. Hematology. 2010 Aug;15(4):215-221. (SCI, IF 1.336, A2, corresponding author )
6.miRNA-21 regulates arsenic-induced anti-leukemia activity in myelogenous cell lines. Med Oncol 2011 Mar;28(1):211-8. Epub 2010 Feb 9. (SCI, IF 2.21, A2, corresponding author)
7.Inhibitory effects of anti-miRNA oligonucleotides (AMOs ) on A549 cell growth. J Drug Target. 2008,16(9 ) 688-693.(SCI,IF 2.491,A2)
8.Prediction of VEGF mRNA antisense oligodeoxynucleotides by RNA Structure software and their effects on HL60 and K562 cells. CELL BIOL INT. 2005,29(9) 737- 741(SCI,IF 1.747)
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